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Description
This study revealed that the tryptophan metabolic pathway was disrupted in NAFLD model rats, resulting in a reduced tryptophan metabolic capacity and significantly upregulated levels of tryptophan
1,5-AG suppresses pro-inflammatory polarization of macrophages and promotes the survival of B-ALL in vitro by upregulating CXCL14
This is informed speculation, not proven protocol
J., Ho E
