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METH-induced cardiotoxicity has been attributed to sympathomimetic stimulation and reactive oxygen species (ROS) production, but several mechanisms are now recognized to be involved (reviewed in Reddy et al
Protective effect of erdosteine on erythrocyte deformability in a rat model of lower limb ischemia/reperfusion injury
Similar to CXCL12CXCR4, which has been proven to take part in PCa bone metastases, CXCL16CXCR6 is expressed in human PCa and bone specimens [227]
Journal of Cosmetic Dermatology , 20(3), 893900
