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Overexpression of miR-146a and miR-200b downregulates NR2A and NR2B proteins, contributing to cognitive deficits [71], while miR-137 controls excitatory synapse maturation and plasticity by regulating GluA1 expression, with its levels modulated by mGluR5 activation [63]

In this review, we summarize the roles and mechanisms of HDACs and SIRTs in kidney diseases and then review the potential therapeutic effects of some SIRT activators and HDAC inhibitors in kidney protection