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Collectively, our data suggest that the increased susceptibility to undergo spontaneous cell death in DADA2 is cell-intrinsic and not mediated by the inflammatory milieu of ADA2-deficient cells and cannot be rescued by inhibition of TNF-alpha, type I interferon signalling or STING signalling
Bohme-Schafer I, Lorentz S, Bosserhoff AK (2022) Role of amino acid transporter SNAT1/SLC38A1 in human melanoma
