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Description
Our data revealed that HuR has the most decisive impact on the KGA/GAC protein level on a cell background that is glutamine withdraw and glutaminase-inhibition resistant (which even includes some TN tumor cells), implying that it is not the HuR levels by itself, but glutamine-dependent TCA cycle itself is the critical factor behind HuR having the most pronounced effect over KGA/GAC levels

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2012;108:S315S332

For instance, a 13% difference in toxinotype 0 and III in C-terminal region of toxin B is important for antibody binding [160,161]
