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It is caused by development of endometrial-like glands and stroma outside the uterus, and though its exact pathogenesis remains unclear, it appears to involve a combination of contributing factors such as retrograde menstruation into the peritoneal cavity 2 , and an altered immune response 3,4

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Moreover, the levels of proteins associated with cell proliferation, migration, and angiogenesis (Slc38a2, Psph, Jagn1, and Rorb) were upregulated
