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doi: 10.1024/0300-9831.74.3.223
While the ipsilateral precursory lesion could be explained by an early focal invasion by immune cells that remain dormant for years, the contralateral mirror lesions are harder to ascribe to such a mechanism
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They demonstrated that mice deficient in VIP or VPAC 1 have reduced numbers of intestinal ILC3s and correspondingly impaired production of IL-22, which, as Seillet et al
