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Park Y, Figueroa ME, Rozek LS, Sartor MA
633635 Studies have demonstrated that CLR01 not only prevents the initial formation of amyloid fibrils but also remodels existing fibrils into nontoxic, off-pathway oligomers
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HDL proteins (apolipoprotein M, apoM) and apoM-binding lipids (S1P) contribute to HDLs ability to increase tight junctions and endothelial cell survival (843)
