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An increased mitochondrial respiratory activity in the liver therefore speeds up the degradation and prevents the accumulation of fatty acids in the liver, which prevents the development of fibrosis in MASH [15]

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coli (Holmgren, 1976) where they have important functions as electron donors for ribonucleotide reductase (RNR), adenosine-5-phosphosulfate (APS) reductase, 3-phosphoadenosine-5-phosphosulfate (PAPS) reductase and arsenate reductases (Holmgren, 1981