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Description
Pancreatic ductal adenocarcinoma (PDAC) Clinically, Overexpression of CEBPB in PDAC tissues correlates with reduced patient survival and aggressive tumor behavior, highlighting its potential as a prognostic biomarker [57, 58]
The expression of ATGL is stimulated by various factors, including proliferator-activated receptor (PPAR) agonists, mammalian target of rapamycin complex 1 (mTORC1) inhibition, and forkhead box O 1 (FOXO1) activation [77,78,79]
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