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Description
Targeting ferroptosis for the treatment of cognitive impairment Given the core pathogenic role of ferroptosis in various cognitive disorders, targeting this pathway has emerged as a highly attractive and novel therapeutic strategy

For example, during iron deficiency, iron regulatory proteins (IRPs, see below) can promote increased TfR expression by stabilizing TfR mRNA [64]

Laboratory investigations demonstrate that GHK-Cu application in experimental tissue models can reverse many of these age-related changes through direct effects on dermal architecture and cellular function
These ChIP-seq and biochemical results collectively suggest that condensin co-localizes with mediator and Tbp1 at gene regions through protein-protein interactions
