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In the POLLUX clinical trial, patients with relapsed or refractory multiple myeloma who received the combination of the anti-CD38 antibody daratumumab with lenalidomide and dexamethasone had significantly prolonged progression-free survival (PFS) relative to those who received lenalidomide and dexamethasone alone

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Therefore, significant molecular changes during the pre-symptomatic phase caused by TrkB signalling depletion may induce iSPN vulnerability

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