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[DOI] [PubMed] [Google Scholar] 172.Kleschyov A.L., Strand S., Schmitt S., Gottfried D., Skatchkov M., Sjakste N., Daiber A., Umansky V., Munzel T

CD117 Anti-CD117-saporin ADCs have also been studied

The virally induced shedding or downregulation of this receptor can cause the endothelium to start producing more angiotensin 2, eventually leading to more oxidative stress in the endothelium

131 The system, termed LNC-Ted HAKPV, offered three principal therapeutic benefits: (1) promotion of endogenous GLP-2 production to enhance intestinal growth and repair, (2) targeted anti-inflammatory action mediated through HA-KPV's modulation of the CD44/TLR4 pathways, and (3) redox-responsive release of the anti-inflammatory tripeptide KPV via disulfide bonds, activated under inflamed intestinal conditions