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Emerging evidence from related myeloid and lymphoid malignancies suggests that such epigenetic insufficiency generates synthetic-lethal interactions with constraints on one-carbon metabolism - including MAT2A-mediated S-adenosylmethionine production - a mechanistic paradigm that warrants prospective investigation in EZH2-mutant AML as a candidate metabolic vulnerability [11]

Kataria, A., Magoon, S., Makkar, B

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