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Description
The four M8-03 mutants were unable to restore the doxorubicin resistance observed in FlpIn 293 cells overexpressing wild-type MRP1 (Supplementary Fig
The authors suggest that glycolysis or other ways of ATP production may supply sufficient ATP necessary for synaptic activity [41]
Mechanisms of physiological and pathological cardiac hypertrophy
It can act as an enzymatic active center in redox reactions, thereby inhibiting oxidative stress [189, 190]
