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These findings are in line with results obtained by other researchers in two pilot clinical trials, which revealed that topical administration of the TRPM8 agonist (cryosim-3) significantly increased basal tear production and lessened neuropathic pain in DED patients, indicating that the use of TRPM8 agonists could be considered as a potential new therapeutic approach for the treatment of DED-related ocular discomfort [29,30]

Translational research further corroborates the link between a deficiency of LAC in clinical phenotypes of TRD and certain aspects of inflammation [51, 58]

Multitargeted approaches and healthy lifestyle, including the combined administration of antioxidant compounds, antioxidant-rich dietary patterns, intellectual stimulation (formation of new synapses), and physical exercise, are strongly advised and currently under investigation in human studies

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