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doi: 10.3390/biomedicines9050464
Epileptogenic injuries or seizures initiate the synthesis and secretion of proinflammatory molecules such as TNF-, IL-1, and High Mobility Group Box 1 (HMGB1) in glial cells which result in decreasing seizure threshold, which further contributes to seizure precipitation, and recurrence because of rapid changes in glutamate and -aminobutyric acid (GABA) receptor phosphorylation

Modulation of the nuclear factor-kappa B (NF-kB) signalling pathway by glutamine in peritoneal macrophages of a murine model of protein malnutrition

Conclusion Wells Fargos performance highlights a bank in transition, balancing cost efficiency, digital growth, and regulatory recovery
