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Motion sickness: an overview
When administered to neuroblastoma cells during the final seven days of high glucose exposure, neohesperidin effectively reversed protein S-nitrosylation and significantly increased cell proliferation, suggesting its ability to mitigate the detrimental effects of oxidative stress
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It predominantly resides in mitochondria, where it exerts control over mitochondrial acetylation and plays a critical role in regulation of metabolic and antioxidant functions [62]
