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In states of hypercortisolemia (e.g., Cushings syndrome), decreased GH secretion with normal to elevated serum IGF-I levels have been reported (99, 100) ( Table 1

Overexpression of GRX480 represses JA-triggered induction of the defensin, PDF1.2 (Ndamukong et al., 2007) and SA-dependent repression of this marker gene is correlated with changes in glutathione status and opposed by inhibition of glutathione synthesis (Koornneef et al., 2008)
