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The activation of Nrf2 by lutein may be caused by the presence of an electrophilic group that combines with the cysteine residue thiols in Keap1 to cause the Nrf2-Keap1 complex to break down, thereby boosting the production of different cytoprotective and antioxidant proteins and supporting their cardioprotective properties

Against this backdrop, PRLMs have emerged as critical modulators capable of restoring immune cell tumor-clearing functions by reshaping their activities within the TME, interfering with tumor evolution [230]

This indicates that PKD3 evoked PKA activity regulates a broader spectrum of hepatic metabolism