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(PubMed) Jones W, Li X, Qu ZC, Perriott L, Whitesell RR, May JM
VCN managed to halt the progression of fibrosis either directly by interfering with the classical TGF-1/Smad 2/3 signaling pathway or indirectly by upregulating the expression of Nrf2, enhancing the antioxidant system, activating PPAR- and downregulating the NLRP3/NF-B pathway which influences the production of inflammatory cytokines involved in the pathogenesis of pulmonary fibrosis
For the remaining 63 spectra, mean fit error was 38%
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