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Taken together, the experimental findings discussed above reveal a complex interplay between alcohol-induced zinc deficiency and two fundamental signaling pathways that are required for strong host immune capacity within the alveolar macrophages

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Gastrodin: Modulating the xCT/GPX4 and ACSL4/LPCAT3 pathways to inhibit ferroptosis after ischemic stroke

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The injured liver, predominantly Kupffer cells and infiltrating macrophages and neutrophils, produces TNF-, interleukin-1 and interleukin-6

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