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These results suggest that the putatively vesicular copper released from neurons following NMDAR stimulation (Schlief et al., 2005) may act on NMDARs themselves to rein in excessive excitation, and that additional copper release can protect neurons from excitotoxic cell death

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Evaluation of 14-3-3 Protein Family Levels and Associated Receptor Expression of Estrogen and Progesterone in Human Uterine Leiomyomas
