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As clinical trials of cell therapies for intervertebral disc degeneration gain increasing attention, a comprehensive assessment of the biological elements of degeneration and regeneration has become particularly essential
In the last years, approaches to design future therapies consider novel aspects as, for example, heritability of GSH levels in different organs/tissues [190], regulation of epigenetic mechanisms as response to variations of glutathione level or GSH/GSSG ration [191]
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