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Histological evidence has shown that the activated T cells cross the BBB, invade the CNS, and congregate around small venules to trigger localized inflammation (Kuhlmann et al., 2017)

While treatment of hepatocytes with phenolic antioxidants induce binding of Nrf2/JunD/Maf to human GCLCs ARE4 to trans -activate the gene, it appears that TGF-1 treatment in type II alveolar cells induced binding of Fra-1/c-Jun dimer to the internal AP-1 sequence of the ARE4 site and suppressed GCLC expression (Jardine et al., 2002)

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Notably, many sdAbs binding to epitope 2 and 3 interfered with the binding of daratumumab, with MU375, MU1053 and MU551 showing competitive binding