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In vitro, the mutant MOB1-K11R, which lacks acetylation, enhances the proliferation, migration and invasion of lung cancer cells, and in vivo, it accelerates tumor growth compared to wild-type MOB1 (105)

Recent findings demonstrating the key role of fatty acid oxidation for renal cell proliferation and compensatory hypertrophy may help to understand the complex mechanisms involved in this process and whether abnormal lipid metabolism in the injured kidney may contribute to IGF-1 resistance

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Notably, these samples were grown until saturation in the absence of Zn before re-testing