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However, dysregulated astrocyte response or high levels of pro-inflammatory cytokines can be harmful to ischemic recovery by increasing the level of toxic nitric oxide and/or inducing neuronal apoptosis directly (Giffard and Swanson, 2005
10.1097/SPC.0000000000000515 191 McQuadeR
Multiple variants across multiple pathways means you can be debilitated by exposures that others around you do not notice
Recent studies have shown that an overabundance of inflammatory cytokines, notably IFN-, and the ensuing activation of the JAK-STAT pathway, could constitute a potential molecular mechanism driving the pathophysiology of sarcoidosis
