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Proc Natl Acad Sci USA 1984;81:132731 [DOI] [PMC free article] [PubMed] [Google Scholar] 9.Leeming MG, Gamon LF, Wille U, Donald WA, OHair RA.What are the potential sites of protein arylation by N-acetyl-p-benzoquinone imine (NAPQI)

Also, mice did not develop ALS-like symptoms upon expression of A4V-mutant human SOD1, but co-expression of wild-type human SOD1 in the A4V-SOD1 expressing mice did trigger the progression of ALS-like disease [29]

Interestingly, pyruvate kinase liver and red blood cell (PKLR) expression is inhibited in liver-specific Keap1 -null mice [82]

Mechanistically, the protection and antiviral effects of these bioactive compounds include a decrease in oxidative damage, inhibition of viral binding and replication, RNA synthesis, caspase enzymes, viral proteases, and viral assembly