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One key metabolic feature of M2 TAMs is lipid metabolism, whereby TAMs enhance lipid uptake and storage through scavenger receptors (CD36, MARCO), fueling mitochondrial respiration and supporting M2 gene expression [34, 35]

In Gclc -KO liver, the GSH level is less than 5%, but in Chac1 -overexpression mice, the liver GSH level is approximately 15%, which is sufficient to maintain normal liver function and mitochondrial respiration (14)

Binding and activation of the seven-transmembrane estrogen receptor GPR30 by environmental estrogens: a potential novel mechanism of endocrine disruption