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Mice harboring a podocyte-specific loss of function knockout of Pdss2 reproduced a more severe phenotype of the Pdss2 kd/kd mice, indicating that the kidney disease in the Pdss2 kd/kd mouse results from the CoQ deficiency in the podocytes [193]

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These compounds included 1-methoxy-2-propanol (Val), 2-methyl-1-butanol (Ile/Leu), 3-methyl-2-butenal (Ile/Leu), 3-methyl-butanoic acid (Leu), 2-methyl-propanoic acid (Val), and sulfur-containing compounds (Cys/Met), which can considerably influence the organoleptic quality of the product (Settachaimongkon et al

10.1007/s00702-018-1934-9 159 MartinL.LatypovaX.WilsonC