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Moreover, metabolic reprogramming in immune cells, particularly mitochondrial dysfunction, has been linked to enhanced immune responses and tissue damage in SLE, further highlighting the role of hypoxia in driving autoimmune pathology (149)
doi: 10.1104/PP.106.077073 4 Pham-HuyLAHeHPham-HuyC
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Consequently, this results in an upregulation of Bcl-2 mRNA expression, a downregulation of Bax mRNA expression, and a reduction in downstream inflammatory factors, ultimately leading to the inhibition of inflammation (122)
