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Both PAD and tTG are active in joints and the intestine, with the latter of these modification proteins being over expressed in the synovium of RA patients suggesting a possible role in disease pathogenesis (Lauzier et al

Additionally, there is an upsurge in nitric oxide synthesis, activation of nuclear factor-B, an increase in nerve fiber density, elevated levels of serum C-reactive protein, and enhanced production of neuropeptide Y and nerve growth factor

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