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caldus adapting to the high osmolarity (Chen et al., A
10.1093/cvr/cvab046 76 Guarner-LansV.Ramrez-HigueraA.Rubio-RuizM
These exposures act via a mechanism involving hormonal dysregulation, oxidative stress buildup, DNA damage, and epigenetic alterations
Recent studies have shown that an overabundance of inflammatory cytokines, notably IFN-, and the ensuing activation of the JAK-STAT pathway, could constitute a potential molecular mechanism driving the pathophysiology of sarcoidosis
