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Disappointing in vivo effects of anticancer molecules mediating strong effects in vitro through IGF-1R silencing were observed, possibly due to cancer cell resistance mediated by compensatory mechanisms activating IGF-1R downstream targets
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ozone: science & engineering, 32:8190, Shema R, Kulicke R, Cowley GS et al (2015) Synthetic lethal screening in the mammalian central nervous system identifies Gpx6 as a modulator of Huntingtons disease
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