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Given the evidence that NMDARs are expressed in the heart and control cardiac excitation and conduction, the question as to their physiological agonist is obviously raised
10.3390/cancers14143538 136 SvensonK
Depletion of mitoNEET leads to mitochondrial iron accumulation and the generation of mitochondrial lipid peroxides contributing to ferroptosis [85, 86]
The other report from that year is the one from Hou et al
