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Thus, in some tumor cells, doxorubicin-induced apoptosis apparently does not rely on oxidant generation, as excess GPx-1 overexpression fails to protect against apoptosis (379)

Besides cytoplasmic action of mGPx4, mGPx4 might act on mitochondrial events as well because mitochondrial ROS or mitochondrial peroxidized lipid accumulated in erastin-treated SK-Hep1 + cells and mitochondrial ROS quenchers inhibited erastin-induced SK-Hep1 + cell death together with abrogated mitochondrial ROS or mitochondrial peroxidized lipid accumulation

Catalyzed by glutathione synthetase ( GSS ), this step is generally not rate-limiting under normal conditions but GSS variants that reduce enzyme activity can become limiting when step 1 is driving high -GluCys throughput, or when glycine availability is restricted

Priming approaches to improve the efficacy of mesenchymal stromal cell-based therapies