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The immune response to this damage, characterized by M1 macrophage polarization, neutrophilic inflammation, and Th17 activation, further exacerbates lipid peroxidation and ferroptosis, creating a vicious cycle that drives disease progression

A study conducted in bleomycin-induced pulmonary fibrosis mice, showed marked deposition of iron and elevated levels of ferroptosis markers (Cheng et al., 2021)

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Regarding the HER2 target, a promising strategy to enhance the persistence of adoptively transferred T cells involves expressing CARs in virus-specific T cells (VSTs) targeting cytomegalovirus, Epstein-Barr virus, or adenovirus, which are genetically modified to express HER2-CARs with a CD28.-signaling endodomain