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This is because active folate (methylfolate) is required for B12-dependent reactions, and MTHFR variants can reduce folate availability

When tissue folate and cobalamin stores are intact, an abnormal increment of postmethionine-load homocysteinemia may also reveal a saturation of the transsulfuration pathway caused by dietary methionine excess that might be combined with transsulfuration defects (in cases with basal hyperhomocysteinemia) or with the homozygous thermolabile MTHFR defect (in cases with normal fasting homocysteine) (Cattaneo, 1999)

[DOI] [PubMed] [Google Scholar] 67.Strinic D., Belosic Halle Z., Luetic K., Nedic A., Petrovic I., Sucic M., Zivanovic Posilovic G., Balenovic D., Strbe S., Udovicic M., et al

Laboratory Tests During the initial treatment of patients with pernicious anemia, serum potassium must be observed closely the first 48 hours and potassium replaced if necessary