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In response to oxidative stress and ionizing radiation, DNA damage can lead to activation of NF-B through p53, potentially inducing pro-apoptotic responses
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(2015), it was shown in a targeted analysis (Mason et al., 2016a) that the highly elevated amounts of lactate observed in the CSF of confirmed TBM cases was only in the L-form (L-lactate is produced by the host, whereas D-lactate is of bacterial origin)
