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In apoptosis, -sitosterol upregulates pro-apoptotic factors such as p53 and Bax, while downregulating anti-apoptotic proteins such as Bcl-2, leading to an increase in mitochondrial outer membrane permeability and the activation of key apoptotic enzyme cascades such as caspase-9 and 3, which induces programmed cell death (83)

PLoS ONE 7:e37709

The first difference is that in the integrative model of oxidative stress and C1 metabolism, formaldehyde accumulation begins nearly 5 times sooner at ~180,000 seconds (~2 days) versus at ~650,000 seconds (~7.5 days)

Microbial components and effector molecules in T helper cell differentiation and function