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In a separate analysis of ApoE / mice on an atherogenic diet for 12 weeks, GPx1 mRNA (detected by in situ hybridization) and GPx1 protein (detected by immunostaining) were highly concentrated in macrophage- and smooth muscle cell (SMC)-rich areas of atherosclerotic lesions of ApoE / aortas [80]

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Physical inactivity and idiopathic pulmonary embolism in women: prospective study

in the other outcome, the injured tissue continues to deteriorate, and the mechanism of the persisting damage involves gliosis with the loss of support of neurotrophic factors, persistent inflammatory receptor activation, and changes in microglia and astrocytes that continue to release harmful cytokines, which promote neuronal death and axonal injury