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Accordingly, strategies aimed at stabilizing CL metabolism or preventing its oxidative remodelling hold considerable promise as mitochondrion-targeted therapies

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The UPR consists of three main signaling pathways, PERK, IRE-1, and ATF6., that normally bound by a chaperone called Grp78 and are inactive

E2F1 and E2F2 induction in response to DNA damage preserves genomic stability in neuronal cells