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Animal studies show enhanced dopamine and serotonin signaling and improved resilience under stress

GHK-Cus mechanism depends on cumulative tissue signaling rather than peak concentration

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Despite T1D children harbouring greater levels of pro-inflammatory mediators, including interferons and interleukins, in their intestine it has been proposed that such immune profiles are a consequence of disease progression rather than the cause of T1D or its exacerbation (Savilahti et al