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In contact dermatitis and atopic dermatitis, inhibition of 15-LOX inhibits podosome formation in dendritic cells, downregulates the antigen uptake and cell migration capabilities, and contributes to the alleviation of skin inflammation (Figure 2)

205 Additionally, the activation of the TNF receptor-1 (TNF-R1) signaling pathway is involved in the copper-induced extrinsic apoptosis pathway, marked by substantial increases in the mRNA and protein levels of TNF-R1, Fas-associated death domain, TNFR-associated death domain, and cleaved caspase-8

This combination improved hypothalamic inflammation, astrocytosis, microgliosis, and insulin sensitivity

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