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Consequently, this results in an upregulation of Bcl-2 mRNA expression, a downregulation of Bax mRNA expression, and a reduction in downstream inflammatory factors, ultimately leading to the inhibition of inflammation (122)
HGSC and CCC are highly sensitive to cystine-deprived death, exhibiting a cystine addiction phenotype [15,34,35]
Journal of Neurotrauma 22(9):1003-1010
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