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Description
Within the process of SCI, the pathological milieu is marked by oxidative stress and inflammation, both of which can intensify neuronal damage and impede recovery
In the latter process, stress-induced activation of the GC-inducible molecule, TSC22D3 [glucocorticoid-induced leucine zipper (GILZ)] in tumour-infiltrating DCs, which inhibited type I interferon (IFN) responses in those cells and IFN- + T-cell activation, was suggested as a possible mechanism [24, 183]
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