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Description
Neuropathological hallmarks of the disease include the abnormal deposition of amyloid -peptide (A) fibrils, the main component of senile plaques (SPs), and the intracellular accumulation of neurofibrillary tangles (NFTs), composed of hyperphosphorylated Tau protein (pTau)
often described as the cells energy centers

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Although subsequent normalization in the blood arterial ammonia level was observed within 4 days, the patient remained comatose for 3 weeks
