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Description
Bauer G (2015) Increasing the endogenous NO level causes catalase inactivation and reactivation of intercellular apoptosis signaling specifically in tumor cells
69) Mol AJ, Voshaar RO, Gorgels WJ, Breteler MH, Van Balkom AJ, Van de Lisdonk EH, Kan CC, Mulder J, Zitman FG
BPC 157 treatment led to the upregulation of Egr1, Akt1, Kras, Src, Foxo, Srf, Vegfr2, Nos3, and Nos1 and the downregulation of Nos2 and Nfkb compared with untreated rats
In other words, hypoxia-responsive NDCDS is stable in the normal physiological environment but is activated once extravasation into the hypoxic tumor microenvironment
