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arsenic trioxide complex glutathione trioxide: Mechanisms of action Mechanism of arsenic toxicity: Arsenic
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[DOI] [PubMed] [Google Scholar] Palta P, Samuel LJ, Miller ER, 3rd, Szanton SL

Nguyen, K., Dersnah, G

When the cellular demand for NAD+ to sustain oxidation exceeds the rate of ATP turnover, particularly in cells exhibiting active aerobic glycolysis, NAD+ regeneration becomes a limiting factor under conditions of compromised mitochondrial respiration

They mimic the brain's own growth factors to support neuron survival
