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CIL and RL lesions shared additional pathways, including TGF and PI3K/AKT/mTOR signaling, suggesting acquisition of lesion-stage-specific remodeling programs
doi: 10.1002/alz.12282
As 2 O 3 induces ROS generation by disrupting the mitochondrial ETC, impairing mitochondrial membrane potentials, and depleting GSH in HeLa and Calu-6 cancer cells [190, 191]
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